Authors

Diana Crowley, Philadelphia College of Osteopathic Medicine
Samantha Murad, Philadelphia College of Osteopathic Medicine
Courtney Helm, Philadelphia College of Osteopathic Medicine
Rachel Souza, Philadelphia College of Osteopathic Medicine
Sarah Coughlan, Philadelphia College of Osteopathic Medicine
Scott Serpico, Philadelphia College of Osteopathic Medicine
Eric Sugarman, Philadelphia College of Osteopathic Medicine
Kyle Marguiles, Philadelphia College of Osteopathic Medicine
Brian Heist, Philadelphia College of Osteopathic Medicine
Kathryn D. Mitchell, Philadelphia College of Osteopathic Medicine
Christopher K. Sutera, Philadelphia College of Osteopathic Medicine
Mark Martin, Philadelphia College of Osteopathic Medicine
Carlos Font, Philadelphia College of Osteopathic Medicine
Mary Woodruff, Philadelphia College of Osteopathic Medicine
E-Jine Tsai, Philadelphia College of Osteopathic Medicine
Rushil Brahmbhatt, Philadelphia College of Osteopathic Medicine
Paul Lecker, Philadelphia College of Osteopathic Medicine
Grzegorz Gorski, Philadelphia College of Osteopathic Medicine
John Benalcazar, Philadelphia College of Osteopathic Medicine
Serena Young, Philadelphia College of Osteopathic Medicine
Abey Martin, Philadelphia College of Osteopathic Medicine
Lindsay Gugerty, Philadelphia College of Osteopathic Medicine
Jacquelyn Gerhart, Philadelphia College of Osteopathic MedicineFollow
Mindy George-Weinstein, Philadelphia College of Osteopathic MedicineFollow
Arturo Bravo Nuevo, Philadelphia College of Osteopathic MedicineFollow

Document Type

Article

Publication Date

2025

Abstract

Myo/Nog cells play a pivotal role in ocular development and demonstrate a rapid response to stress and injury. This study investigates their behavior and distribution in a murine model of retinitis pigmentosa, specifically in C3H/HeJ mice, which exhibit photoreceptor degeneration due to a homozygous mutation in the Pde6brd1 gene. Retinal samples from C3H/HeJ and C57BL/6J mice were analyzed at postnatal weeks 2.5 to 6 using hematoxylin and eosin staining, immunofluorescence for brain-specific angiogenesis inhibitor 1 (BAI1) expressed in Myo/Nog cells, and TUNEL labeling for apoptotic cell detection. The results demonstrated a progressive thinning of the outer nuclear layer (ONL) in C3H mice, accompanied by a significant increase in Myo/Nog cell numbers. In normal retinas, Myo/Nog cells were primarily located in the inner nuclear and outer plexiform layers. However, in C3H/HeJ mice, they accumulated in the ONL near apoptotic photoreceptors and within the choroid. Notably, in these degenerative regions, Myo/Nog cells exhibited features of phagocytosis, suggesting a role in apoptotic cell clearance. Additionally, parallels between Myo/Nog cell responses in retinitis pigmentosa and models of oxygen-induced retinopathy, ocular hypertension, and light damage suggest that these cells may be leveraged for therapeutic purposes.

Publication Title

Applied Sciences (Switzerland)

Comments

This article was published in Applied Sciences (Switzerland).

The published version is available at https://doi.org/10.1007/978-3-031-78244-2_68.

Copyright © 2025 by the authors. CC BY 4.0.

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