Polymorphisms of DNA repair genes XPD, XRCC1, and OGG1, and lung adenocarcinoma susceptibility in Chinese population
Document Type
Article
Publication Date
2013
Abstract
Lung adenocarcinoma (ADC) is one of the major histological types of lung cancer. Genetic polymorphism in DNA repair genes and lung ADC susceptibility is well documented. In this case-control study, the association between the polymorphic sites of DNA repair genes XPD-751, XRCC1-399, and OGG1-326, and lung ADC susceptibility in ethnic Han Chinese population has been investigated. Genomic DNA was isolated from the peripheral blood of 201 healthy controls and 82 lung ADC patients from the people of Hunan Province, China. Polymorphisms of the investigated genes were analyzed by using polymerase chain reaction-restriction fragment length polymorphism. There was no significant difference between the samples from lung ADC patients and healthy controls about the genotype frequencies of XPD-751, XRCC1-399, and OGG1-326 sites. However, multifactor dimensionality reduction analysis showed that the genetic polymorphisms of the three-loci models of DNA repair genes (XPD-751/XRCC1-399/ OGG1-326) are associated with lung ADC. Thus, this study reveals that a three-order interaction among the polymorphic sites of XPD-751, XRCC1-399, and OGG1-326 is associated with lung ADC risk in the studied population, although polymorphism in individual gene was not associated. © 2013 International Society of Oncology and BioMarkers (ISOBM).
Publication Title
Tumor Biology
Volume
34
Issue
5
First Page
2843
Last Page
2848
Recommended Citation
Ouyang, Fangdan; Yang, Fulan; Cheng, Hanchun; Khan, Md. Assduzzaman; Huang, Fengmao; Wan, Xinxing; Xu, Aihua; Huang, Xing; Zhou, Meijuan; and Zhang, Dianzheng, "Polymorphisms of DNA repair genes XPD, XRCC1, and OGG1, and lung adenocarcinoma susceptibility in Chinese population" (2013). PCOM Scholarly Works. 311.
https://digitalcommons.pcom.edu/scholarly_papers/311
Comments
This article was published in Tumor Biology, Volume 34, Issue 5, Pages 2843-2848.
The published version is available at 10.1007/s13277-013-0844-6.Copyright © 2013 Springer.